Acute Renal Failure
ACUTE RENAL FAILURE 4
Withregard to the latest laboratory test that was conducted following aseries of medications, the patient’s condition has improvedremarkably, thus he is no longer suffering from acute renalfailure. Primarily, patients with acute renal failure recordoverwhelmingly advanced BUN levels. The blood urine nitrogen levelsof such patients remain consistently high throughout the entireailment period. Once he was admitted to the healthcareinstitution, the patient was recorded to have BUN levels of 81 mg/dl.However, the mentioned blood urine nitrogen level of the patient hadsubstantially reduced to 16 mg/dl. This level is presumed to befairly close to the optimum BUN.
Afterarriving at the hospital, the diagnosis of the patient’s conditioncan be done through ascertaining whether there is a decline in theclearance of creatinine. Laboratory tests should also be conductedsuch tests include osmolality, the microscopy of urine, as well asBUN level. Alcohol consumption is one of the chief risk factors thatelevate the susceptibility of a patient to develop acute renalfailure. Essentially, alcohol affects the functioning of the kidneycells, especially through the development of inflammations. Theinflammations contribute to the blockage of the tubular cells, aswell as the glomerulus (Glynneet al, 2002).Since the patient has a history of alcoholism, it is evident that theconsumption of alcohol has contributed to his condition. Theimplications of the inflammations, among other infections of thekidney cells, are that the kidneys fail to filter blood offimpurities. The impurities accumulate in the blood, and thisaggravates the entire health problem. It is worth noting that age isanother risk factor for acute renal failure. Given the advanced ageof the client, it is apparent that most of the body cells haveprogressively deteriorated over time (Glynneet al, 2002).
The enhancement ofthe electrolyte level owes credit to the fact that the patient hasceased the drinking habit. This means that more and more kidney cellsare being restored to a functional state (Glynneet al, 2002).Further, alcohol poisoning of the kidney cells has also been stoppedfollowing the patient’s admission to the hospital. Coupled withmedication, the patient’s health stabilized significantly leadingto improvement in the electrolyte status.
Glynne,P., Allen, A., & Pusey, C. D. (2002). Acuterenal failure in practice.London: Imperial College Press.